Eliminating senescent cells: As one ages or is affected by chemotherapy, there will be some "senescent cells" in the body that no longer divide but do not die. They will secrete harmful substances and trigger chronic inflammation. Studies have shown that fisetin, as an agent for eliminating senescent cells, can selectively induce the apoptosis of these "zombie cells", thereby reducing their negative impact on the body.

Antioxidation and repair: Compared to common antioxidants, fisetin exhibits stronger free radical clearance ability in computer simulation studies, and can even repair oxidatively damaged biomolecules (such as proteins) and restore their functions.

Several studies have explored the potential role of fisetin in neurodegenerative diseases. It can inhibit inflammatory factors, reduce reactive oxygen species (ROS), and protect neurons from apoptosis through mechanisms such as these, showing potential in improving cognitive function in cell and animal models of Alzheimer's disease and Parkinson's disease. It is believed that it helps alleviate brain inflammation related to aging and supports synaptic function.

Anti-inflammatory: Fisetin can inhibit key pro-inflammatory signaling pathways (such as NF-κB), thereby reducing the production of inflammatory factors such as tumor necrosis factor-α (TNF-α) and interleukin-6 (IL-6).

Anti-cancer: Some studies suggest that fisetin can exert anti-tumor activity by inhibiting specific signaling pathways and inducing apoptosis of cancer cells, but this is mainly at the stage of basic research.